CBD Therapeutic Benefits
The World Health Organization published a report on CBD in November of 2017. Below is an overview of which CBD may have therapeutic benefits.
| Disease | Effects |
| Alzheimer’s disease | Anti-inflammatory, antioxidant, antiapoptotic in vitro and in vivo models
of Aβ-evoked neuro-inflammatory and neurodegenerative responses. |
| Parkinson’s disease | Attenuation of the dopaminergic impairment in vivo; neuroprotection; improvement of psychiatric rating and reduction of agitation, nightmare
and aggressive behavior in patients. |
| Multiple sclerosis | Improved signs of EAE in mice, ant-inflammatory and immunomodulatory
properties. |
| Huntington’s disease | Neuroprotective and antioxidant in mice transgenic models; no significant
clinically important differences in patients. |
| Hypoxia-ischemia injury | Short term neuroprotective effects; inhibition of excitotoxicity, oxidative
stress and inflammation in vitro and rodent models. |
| Pain | Analgesic effect in patients with neuropathic pain resistant to other
treatments. |
| Psychosis | Attenuation of the behavioral and glial changes in animal models of
schizophrenia; anti-psychotic properties on ketamine-induced symptoms |
| Anxiety | Reduction of muscular tension, restlessness, fatigue, problems in concentration, improvement of social interactions in rodent models of
anxiety and stress; reduced social anxiety in patients. |
| Depression | Anti-depressant effect in a genetic rodent model of depression. |
| Cancer | Antiproliferative and anti-invasive actions in a large range of cancer types;
induction of autophagy-mediated cancer cell death; chemopreventive effects. |
| Nausea | Suppression of nausea and conditioned gaping in rats |
| Inflammatory diseases | Ant inflammatory properties in several in vitro and in vivo models;
inhibition of inflammatory cytokines and pathways. |
| Rheumatoid arthritis | Inhibition of TNF-α in an animal model |
| Infection | Activity against methicillin-resistant Staphylococcus aureus |
| Inflammatory bowel and Crohn’s diseases | Inhibition of macrophage recruitment and TNF-α secretion in vivo and ex vivo reduces disease activity index in Crohn’s patients. |
| Cardiovascular diseases | Reduced infarct size through antioxidant and anti-inflammatory
properties in vitro and in vivo. |
| Diabetic complications | Attenuation of fibrosis and myocardial dysfunction |
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